Anti-Aging & Longevity Research Peptides

The biology of ageing is increasingly understood at a molecular level — telomere shortening, mitochondrial dysfunction, cellular senescence, and inflammaging. A new generation of peptide research is targeting each of these mechanisms specifically. QSC supplies the most comprehensively studied longevity compounds at ≥99% HPLC purity.

Epitalon (Epitalon)

CAS 307297-39-8 · MW 390.35 Da · Ala-Glu-Asp-Gly tetrapeptide

Epitalon is a synthetic tetrapeptide originally derived from pineal gland peptide research by the St. Petersburg Institute of Bioregulation and Gerontology. It is studied for its ability to stimulate telomerase activity — the enzyme responsible for maintaining telomere length at chromosome ends. Telomere shortening is a key biomarker and likely mediator of cellular ageing. Preclinical studies have demonstrated Epitalon-associated telomere elongation, antioxidant effects, and melatonin regulation in animal models.

GHK-Cu (Copper Peptide)

CAS 89030-95-5 · MW 340.38 Da · Gly-His-Lys tripeptide + copper

GHK-Cu is a naturally occurring copper-binding tripeptide found in human plasma, saliva, and urine. Plasma concentrations decline significantly with age (from ~200ng/mL at age 20 to ~80ng/mL at age 60), paralleling the decline in wound healing capacity and skin collagen content. GHK-Cu is studied for collagen synthesis stimulation, angiogenesis, anti-inflammatory activity, and nerve growth factor (NGF) upregulation. Its role in gene expression regulation — including VEGF, FGF, and MMP modulation — makes it one of the most broadly researched peptides in the longevity and cosmetics science literature.

NAD+ (Nicotinamide Adenine Dinucleotide)

CAS 53-84-9 · MW 663.43 Da · Coenzyme

NAD+ is the master coenzyme of cellular energy metabolism and a critical cofactor for sirtuin proteins (SIRT1-7), which regulate gene expression, DNA repair, and metabolic adaptation. NAD+ levels decline approximately 50% between ages 20 and 50 in human tissue, with consequences for mitochondrial function, inflammatory signalling, and circadian rhythm regulation. Direct NAD+ supplementation via IV or subcutaneous administration bypasses the rate-limiting NAD+ biosynthesis pathway, making it a focus of longevity researchers seeking to restore youthful NAD+ levels.

Longevity Compound Overview